Supplemental Table 2: Expression levels of genes that are potentially involved in the maintenance of genomic stability.

Table 2. Commonly differentially expressed genes for the comparisons of diploid genomically stable (dGS)  aneuploid genomically stable (aGS)  and aneuploid genomically unstable (aGU) breast carcinomas.

Supplementary Table 1. Profile genes associated with overall survival.

Table 2 Forty genes with validated differential expression between primary breast and metastatic lymph node tumors; Genes upregulated in lymph node metastasis

Table 2 Forty genes with validated differential expression between primary breast and metastatic lymph node tumors; Genes downregulated in lymph node metastasis

TABLE 4 Genes downregulated (P < 0.05 and fold change cutoff < 0.6) in small intestinal epithelial cells of mice consuming sucrose- vs. cornstarch-containing diets

Genes upregulated (P < 0.05 and fold change cutoff > 1.5) in small intestinal epithelial cells of mice consuming sucrose- vs. cornstarch-containing diets

Supplementary Data 5 NSCLC survival associated genes. The set of 72 prognostic classifier genes is ranked according to the gene expression association with recurrence-free survival.

Table 2. Comparison among gene expression profiles of the C57BL/6J  DBA/2  and ICR

Table S2. Levene's test for equal variance within strains on control and smoke exposed mice

Table S3. T-test for differential expression between control and smoke exposed mice

Table 3. List of susceptible genes induced at the same time in DBA/2 and C57BL/6J strains and inhibited or not modulated in ICR smoke-resistant strain

Table 6. Different signature genes modulated in susceptible strains

Table S1. Shapiro-Wilk's test for normality on control and smoke exposed mice

Table 5. List of susceptible genes in DBA/2 strain

Table 4. List of susceptible genes in C57BL/6J strain

Table 2a Identification of upregulated genes in apoptotic esophageal cancer cells compared with non-apoptotic cells

Table 2b Identification of upregulated genes in apoptotic esophageal cancer cells compared with non-apoptotic cells