Table S3c. AR pathway genes differentially expressed between normal prostate  low- and highgrade PC and metastases.

Table S3b. AR pathway genes differentially expressed between normal prostate  low- and highgrade PC and metastases.

Suppl Table S1a R1881 up-regulated spots in LNCaP

Table S3a. AR pathway genes differentially expressed between normal prostate  low- and highgrade PC and metastases.

Table 1. Up-regulated genes identified by intuitive selection

Supplementary Table 2. Genes with Altered Expression in Cleaved vs. Native Antithrombin-Treated HUVECs

Table 1. Effects of bFGF on the changes in HUVEC gene expression induced by cleaved antithrombin

Supplementary Table 5. Statistically identified genes  whose expression was decreased due to copy number losses in the primary tumors.

Supplementary Table 3. Statistically identified genes  whose expression was increased due to copy number gains in the primary tumors.

Table 2. Statistically identified genes  whose expression was increased due to copy number gain both in the cell lines and primary tumors

Supplementary Table 4. Statistically identified genes  whose expression was decreased due to copy number losses in the cell lines.

Supplementary Table 2. Statistically identified genes  whose expression was increased due to copy number gains in the cell lines.

Table 3. Genes up- or downregulated more than 10-fold in the smoke?LPS group

Table 2. Genes up- or downregulated more than 10-fold in the LPS group

Figure 3: Hierarchical clustering of genes and treatment conditions. The sum of differentially expressed genes in three treatment groups (940 genes) was used for the analysis

Table 1. Genes up- or downregulated more than 10-fold in the smoke group

SuppTable1. Gene List for liver metastasis

Supplementary Table 2. Genes with highest discriminatory power (n=128) according to the SVM-RFE method discriminating CML patients with different primary cytogenetic response.

54-gene set for discrimination between luminal A and basal-like subtypes

Table 1 Categorical list of differentially expressed genes in benign stromal fibroblasts treated with conditioned media from malignant breast epithelial cells  vs. normal breast epithelial cells to investigate paracrine interactions between adipose fibroblasts and malignant epithelial cells are essential for structural and hormonal support of breast tumorsParacrine interactions between adipose fibroblasts and malignant epithelial cells are essential for structural and hormonal support of breast tumors