Table 1. Genes upregulated by more than fivefold in CD133+ CB and PB cells compared with CD133- cells

Supplemental Table 7. Genes down-regulated by more than 2 fold in CD133+ as compared to CD133- cells

Table 1b. Decrease in gene expression in T98 cells by IFN-?

Table1a. Ten most differentially regulated plasma cell signature genes in PCT and LBL from iMycEA mice

Table1b. Ten most differentially regulated plasma cell signature genes in PCT and LBL from iMycEA mice

Table1c. Ten most differentially regulated plasma cell signature genes in PCT and LBL from iMycEA mice

Table 2. Examples of genes modulated by ATRA and ATRA plus NSC682994

Fig. 1. The p53 signature is associated with p53 status in independent datasets. Clustergrams are oriented as outlined in Fig. 5. (A) Expression profiles of the Uppsala tumors segregated by the 32-gene signature. Unigene symbols and GenBank IDs are listed to the right.

Table 2. p53 status -associated genes by SAM

Table 3. MMR(mismatch repair)-Status Signature Genes: 100 Genes Found to Be Differentially Expressed in MMR(+) and MMR(-) Proximal Colon Cancers

Table 4. MMR-Related Epithelial Cell Signature Genes

Table 4. Twenty-five most significant probe sets representing 20 genes identified by MAS 5.0  dChip  and RMA

Table 1. Top 33 genes associated with erlotinib treatment in EGFR-negative tumors

Table 3. Genes differentially expressed betweenprimary chemoresistant and primary chemosensitive ovarian cancers (P < 0.001)

Table S6. Transcripts Upregulated in ST-HSC Compared to MPP

Table S2. Transcripts Upregulated in LT-HSC Compared to ST-HSC

Table S5. Transcripts Upregulated in MPP Compared to ST-HSC

Table S3. Transcripts Upregulated in MPP Compared to LT-HSC

Table S1. Transcripts Upregulated in ST-HSC Compared to LT-HSC

Table S4. Transcripts Upregulated in LT-HSC Compared to MPP

Table I. Consolidated List of Genes Demonstrating Significant Up-Regulation (>1.75) or Down-Regulation (<0.5) in the DU145 Cell Line Following 12 or 24 hr Exposure to Saha (1.0 M)

Table 1. Genes whose expression is altered in vitro and in vivo following activation of HGF/Met signaling