Table 2. Top 31 Differentially Expressed Probe Sets by Unequal-Variance t Test (n = 82  false discovery rate  0.5%)

Supplementary Table 2. Genes in the metagenes selected for predicting recurrence. The genes contained in the top 9 metagenes selected in the tree model analyses together with Gene Ontology (GO) annotations.

Supplementary Table 4. Annotations and expression intensities for the probe sets in Fig. 4a.

Supplementary Table 2. Annotations and expression intensities for the probe sets in Fig. 1c.

Supplementary Table 3. Annotations and expression intensities for the probe sets in Fig. 2b.

Table 1. Genes differentially expressed in response to E2F3 knockdown in 5637 bladder cancer cells

Table 2. Differentially expressed genes identified in N-ras knockout cells

Table 1. Differentially expressed genes identified in H-ras knockout cells

Table 3. Differentially expressed genes identified in H-ras -/-/N-ras -/- knockout cells

Supplementary Table 3a: Genes whose expression is upregulated in tumors

Supplementary Table 3b: Genes whose expression is downregulated in tumors

Table 2. Differentially expressed genes with biological function between high- and low-risk patients

Table 3. 45 genes up-regulated in tumoral tissue  comparing normal mucosa to matched tumor colon tissue.

Table 4. 47 genes down-regulated in tumoral tissue  comparing normal mucosa to matched tumor colon tissue.\\ 16919171-Table4.txt\""

top 70 genes with highest levels of consistent correlation with tFA in 3 cancer associated data sets

Table 3. Partial expression data of miRNA clusters in six leukemia cell lines

Table 1. Lung nodule diagnostic genes; eight chosen for further study for each tissue type  four genes expressed at relatively higher levels in a single tissue type  and four genes expressed at relatively higher levels in an equal number of all other tissue types combined

Table 1 the 70-gene signature (the ?Coxranked? signature)  which maximized the HR

TABLE II-B. Representative list of up-regulated genes in biliary epithelial cells of chronic hepatitis C  in comparison with levels in normal subjects