Table 8 Genes uniquely expressed in acutely HTLV-1 transformed B1 and C5 cells

Table 6 Deregulation of DNA repair proteins in acutely HTLV-1 transformed cells

Table 5 Deregulation of TNFR-mediated apoptotic pathways in acutely HTLV-1 transformed cells

Table 7 Deregulation of cell cycle machinery in acutely HTLV-1 transformed cells

Table 1. Microarray analysis of mRNA expression in a comparison of FRS-transfected cells and control

Table 2. Genes used in tree and linear discriminant models of short-term versus long-term survival

Table 2d. List of upregulated genes after hypoxic selection that are known to be involved in the cellular response to oxidative stress and apoptosis signalling

Table 2c. List of hypoxia-inducible genes which are downregulated after hypoxic selection

Table 2a. List of the 25 genes with the strongest upregulation after hypoxic selection

Table 2b. List of the 25 genes with the strongest downregulation after hypoxic selection

Table 2. Differentially expressed genes in PDAC exclusively identified by meta-analysis

Table 2. Common stress response genes (H-ESR genes)

Breast Cancer Molecular Subtype Geneset from Figure 2A . 269 nonredundant Affy U133A probesets that map to the Stanford 'intrinsic' molecular subtypes of breast cancer gene list  (Perou et al.  2000). GeneList was obtained from Cluster Data is Supplementary Data Sheet 5

most discriminant genes by Wilcoxon test for each pairwise comparison in our data (luminal vs basal  basal vs apocrine  apocrine vs luminal)

SuppTable 3. List of reporter genes used in our classifier for the ?ER+  ER/age high? group.

SuppTable 1. List of analyzed genes.  The microarrays included 499 CpG sites from 117 genes.

Table 3. Fold changes of differentially expressed genes in a dye-swap experiment using pooled NN and MM samples

Table 7. Genes Whose Expression Is Preferentially Regulated in the Testis/Epididymis  as Compared to the Uterus/Ovaries  by Transplacental Exposure to 17 alpha-Ethynyl Estradiol

Table 3. Partial List of Genes Whose Expression Is Significantly Regulated by Genistein (Ges)