Table 8. Genes Whose Expression Is Preferentially Regulated in the Uterus/Ovaries  as Compared to the Testis/Epididymis  by Transplacental Exposure to 17 alpha-Ethynyl Estradiol

Table 4. Partial List of Genes Whose Expression Is Significantly Regulated by Bisphenol A (BPA)

Table 6. Genes Whose Expression Is Regulated by Transplacental Exposure to 17 alpha-Ethynyl Estradiol in Both Testis/Epididymis and in the Uterus/Ovaries  in the Same Direction

Table 5. Gene Expression Changes Induced by Transplacental Exposure to EE  Ges  or BPA in the Fetal Testis/Epididymis

Table 2. Partial List of Genes Whose Expression Is Significantly Regulated by 17 alpha-Ethynyl Estradiol (EE)

Table S1a Genomic gains and losses influence expression levels of genes located within the affected regions: a study on acute myeloid leukemias with trisomy 8

Table S1e Genomic gains and losses influence expression levels of genes located within the affected regions: a study on acute myeloid leukemias with deletion 5q

Table S1d Genomic gains and losses influence expression levels of genes located within the affected regions: a study on acute myeloid leukemias with monosomy 7

Table S1b Genomic gains and losses influence expression levels of genes located within the affected regions: a study on acute myeloid leukemias with trisomy 11

Table S1c Genomic gains and losses influence expression levels of genes located within the affected regions: a study on acute myeloid leukemias with trisomy 13

Table 1.Cancer Testis genes and their expression in normal tissues

Table 1. Genes up-regulated under hypoxic conditions identified by microarray expression analysis of wild-type murine fibroblasts. Functions indicated for the genes were derived from genecards

Table 4. Genes up-regulated by hypoxia  analysed for hypoxia-related mediation by HIF-1. Column HIF ?/? vs wt indicates differentially expressed genes in wild-type and HIF ?/? cells; hyp vs norm shows genes regulated by hypoxia in both cell lines; interaction HIF-1/hyp determines whether regulation of the gene by hypoxia is mediated by HIF-1; HIF-1 regulated are genes that show an interaction with HIF-1 under hypoxic conditions (cut-off 0.5/?0.5)