Supplementary Table 1. Differentially expressed genes in A549 and MCF7 cells treated with i6A or untreated (P<0.001).

Supplementary Table 26. Genes driving positive enrichment of hematopoietic stem/progenitor gene set in P9906 high-risk ALL.

Supplementary Table 27. Genes driving negative enrichment of the B-cell signal transduction gene set in P9906 high-risk ALL.

Figure 1-2 Bioinformatic analysis of confirmed genes

Supplementary Table S2A. Representative Genes Highly Expressed in Group A compared to B

Supplementary Table S2B. Representative Genes Highly Expressed in Group B compared to A

Table5 Common deletions B-cell development genes

Table 3. Inflammatory signaling and immune response genes that were down-regulated following estrogen administration

Table S1 Differentially expressed genes showing > 2 fold changes and p<0.01 for 0.01 mM NS-398  0.1mM ibuprofen and COX-2 RNAi.

Table S3 Differentially expressed genes showing > 2 fold changes and p<0.01 for 1.5 mM ibuprofen

Table S2 Differentially expressed genes showing > 2 fold changes and p<0.01 for 0.1 mM NS-398.

Table 2. DAVID analysis identified 23 transcription factors present in our high confidence SOX4 target genes

TableS3 Genes differentially methylated between CEBPAsil cases and normal CD34+ bone marrow cells

TableS5 Genes differentially methylated between CEBPAsil leukemias and a selection of T ALL cases

TableS2 Genes differentially methylated between CEBPAsil and CEBPAmut leukemias

Supplementary table 1a. Up- and down-regulated tags in HCC.

Supplementary table 1b. Up- and down-regulated tags in HCC.

TableS2 19-probe set signature for CEBPA mutation in the context of the entire AML cohort

TableS3 21-probe set signature for CEBPA double-mutation in the context of the entire AML cohort

SuppTable 1: The top 100 genes by SAM score with differential expression between patients with ALI + sepsis and sepsis alone

Supplementary Table 3. Integrated signature

Supplementary Table 4 Transcriptomic signature

Table 1 List of the candidate upregulated genes in FLT3/ITD polyclonal stable 32D cell lines as compared to wild type FLT3 harboring 32D cells and transduction controls.