Molecular profiles of all 61 tumors as defined by the 110 nonredundant genes whose expression statistically significantly differentiates BRCA1 and BRCA2 tumors (P<.0001).

Table 2. Classifiers of immune responsiveness

Table 2. Genes identified in subtractive cDNA library of nematode infection (NeMphi) a model of alternatively activated macrophages

Table II. Genes That Were Up- or Downregulated After Genistein Treatment

Table I ? Upregulated Genes in all Passages of UT-DEC-1 (an HPV-33-positive cell line) Compared to Control Keratinocytes and Average Fold of Upregulation

Table II ? Downregulated Genes in All Passages of UT-DEC-1 (an HPV-33-positive cell line) Compared to Control Keratinocytes and Average Fold of Downregulation

Table III ? Upregulated Genes During Progression From Passage 20 to 172 OF UT-DEC-1 (an HPV-33-positive cell line) and Fold of Upregulation

Table IV ? Downregulated Genes During Progression From Passage 20 to 172 of  UT-DEC-1 ((an HPV-33-positive cell line) and Fold of Downregulation

Table 6. Cytokines  lineage-specific receptors  and transcription factors on the myeloid cDNA microarray

Table 7. Genes up-regulated during ATRA/IL-3–induced EML cell differentiation

Table 8. Genes down-regulated during ATRA/IL-3–induced EML cell differentiation

Table 3. Gene expression monitoring by DNA microarray and Northern blot. Shown are gene products  accession numbers for each sequence  and the ratio of expression values obtained in cancerous versus normal tissue as accessed by microarray analysis (Fig. 2A B) and Northern blot (Fig. 3)

Table 5. Down-regulated genes in liver cancer patients. Shown is a subset of genes with reduced expression in liver cancer patient samples relative to a patient with a normal  healthy liver.

Table 4. Up-regulated genes in liver cancer patients. A subset of genes is shown with elevated expression in liver cancer patient samples relative to a patient with a normal  healthy liver.

Table 2. GenBank Accession Number  Gene Name  and Reported Chromosomal Localization of Gene Transcripts Differentially Expressed Between WHO Grade I and WHO Grades II and III Meningiomas