Figure S2: Differential expression of NFkB target genes in MLBCL vs. DLBCL. The results of the permutation analysis are listed in the corresponding table below.

Figure S4: Differential expression of NFkB target genes in Host Response (HR) DLBCL vs. non-HR DLBCL. The results of the permutation analysis are listed in the corresponding table below.

Table S1b: NFkB (nuclear factor kappa B) Target Gene Sets; Genes included in the Lymphochip data set but not the 15k subset

Table S1a: NFkB (nuclear factor kappa B) Target Gene Sets; Genes included in the subset of 15k genes selected according to a MAD(median absolute deviation)-based variation filter

Supplementary Table 1. Gene List of nonseminomatous male germ cell tumors

Appendix 1 Gene list generated by MaxT testing.

Table 4. Genes with statistically significant upregulated expression in CIN3. Six genes were indicated by all three analysis methods (BRB Array-Tools  SAM  Focus). Fold changes are shown as calculated by SAM.

Table 1. Selected genes upregulated by HDI-BT exposure

Table 2. Selected genes downregulated by HDI-BT exposure

Table 1. Epigenetically Silenced Genes in Ishikawa Cell

Table 1. The differentially expressed genes between A9 and A10 DA neurons (>2-fold  FDR<1%)

Additional File 1b: The fifty most up regulated and down regulated genes from the tumor v donor  tumor v adjacent normal and adjacent normal v donor comparison.

Additional File 1a: The fifty most up regulated and down regulated genes from the tumor v donor  tumor v adjacent normal and adjacent normal v donor comparison.

Additional File 1c: The fifty most up regulated and down regulated genes from the tumor v donor  tumor v adjacent normal and adjacent normal v donor comparison.

Table 1 Genes involved in morphological modifi cation

Table 2 Genes involved in the Wnt signalling pathway and cell growth

Figure S2 Genes show up- or down-progression correlated with the phenotypes

Table 4 Expression NF-jB subunits and activating factors in acutely HTLV-1 transformed cells

Table 3 Expression of apoptotic and anti-apoptotic genes in PHA/IL-2 activated CD4+ T-cells and acutely HTLV-1 transformed CD4+ T-cells